Search Results - "proteins"

Hereditary hemochromatosis what have we learnt from population studies /

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Tolerance and autoimmunity

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Hereditary hemochromatosis what have we learnt from population studies /

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Tolerance and autoimmunity

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Adaptor mediated activation of the Hsp100/ Clp protein ClpC controls general and regulatory proteolysis in Bacillus subtilis

Table of Contents: “…Contents: Adaptor protein activation of ClpCP mediated proteolysis in B. subtilis Hsp100/Clp proteins and proteolysis -- ClpCP of B. subtilis in competence development and protein quality control -- Adaptor protein (MecA) mediated activation of ClpCP -- McsB a new regulated adaptor protein -- Adaptor protein network for ClpCP of B. subtilis.…”
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Adaptor mediated activation of the Hsp100/ Clp protein ClpC controls general and regulatory proteolysis in Bacillus subtilis

Table of Contents: “…Contents: Adaptor protein activation of ClpCP mediated proteolysis in B. subtilis Hsp100/Clp proteins and proteolysis -- ClpCP of B. subtilis in competence development and protein quality control -- Adaptor protein (MecA) mediated activation of ClpCP -- McsB a new regulated adaptor protein -- Adaptor protein network for ClpCP of B. subtilis.…”
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Adaptor mediated activation of the Hsp100/ Clp protein ClpC controls general and regulatory proteolysis in Bacillus subtilis

Table of Contents: “…Contents: Adaptor protein activation of ClpCP mediated proteolysis in B. subtilis Hsp100/Clp proteins and proteolysis -- ClpCP of B. subtilis in competence development and protein quality control -- Adaptor protein (MecA) mediated activation of ClpCP -- McsB a new regulated adaptor protein -- Adaptor protein network for ClpCP of B. subtilis.…”
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The evolution of complement and the alternative pathway

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The evolution of complement and the alternative pathway

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The neuronal ceroid lipofuscinoses

Table of Contents: “…Contents: Clinical features of the neuronal ceroid lipofuscinoses -- Genetics -- Genotype/phenotype correlations -- Protein epidemiology -- Mechanisms of neurodegeneration -- Treatment approaches.…”
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The neuronal ceroid lipofuscinoses

Table of Contents: “…Contents: Clinical features of the neuronal ceroid lipofuscinoses -- Genetics -- Genotype/phenotype correlations -- Protein epidemiology -- Mechanisms of neurodegeneration -- Treatment approaches.…”
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Gene silencing in budding yeast

Table of Contents: “…Contents: Gene silencing: an epigenetic phenomenon -- Chromatin remodeling: heterochromatin and euchromatin -- Silent chromatin in budding yeast: mating-type loci -- Silent information regulation: telomeric genes and Sir proteins.…”
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Gene silencing in budding yeast

Table of Contents: “…Contents: Gene silencing: an epigenetic phenomenon -- Chromatin remodeling: heterochromatin and euchromatin -- Silent chromatin in budding yeast: mating-type loci -- Silent information regulation: telomeric genes and Sir proteins.…”
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Heritable arrhythmias insight into molecular physiology from the long QT syndrome /

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Complement and organ transplantation

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Complement in PNH and other hemolytic anaemias part 1 of 3 /

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CLL novel prognostics, and updates on therapy - part 2 of 2 /

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Microbial recognition and the immune response

Table of Contents: “…-- Overview of innate immunity -- Cell signaling PRMs -- Toll-like receptors -- Shigella flexneri -- NLRs are related to plant R proteins involved in disease resistance -- NLRs: detectors of MAMPs and danger signals -- Proposed mechanism for Nod protein activation by a MAMP -- Alerting the cell: Nod1 activates NFkB -- Critical role of Nod1 in infected intestinal epithelial cells -- Proposed mechanism for NLRP protein activation by a DAMP -- The inflammasomes -- How do innate immune signals translate into adaptive immune responses? …”
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Series (Immune System - key concepts and questions)
Series (Molecular basis of bacterial infection)

Mitochondrial fatty acid oxidation deficiencies

Table of Contents: “…Contents: Mitochondrial fatty acid oxidation (FAO) defects -- FAO biochemical pathways -- FAO defects -- Diagnosis of FAO defects -- Newborn screening for FAO defects, especially medium-chain acyl-CoA dehydrogenase (MCAD) deficiency -- Diagnostic genetic analysis for MCAD gene variations -- Expression studies of variant MCAD proteins in E.coli -- Protein misfolding as a pathogenic mechanism in MCAD deficiency.…”
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Mitochondrial fatty acid oxidation deficiencies

Table of Contents: “…Contents: Mitochondrial fatty acid oxidation (FAO) defects -- FAO biochemical pathways -- FAO defects -- Diagnosis of FAO defects -- Newborn screening for FAO defects, especially medium-chain acyl-CoA dehydrogenase (MCAD) deficiency -- Diagnostic genetic analysis for MCAD gene variations -- Expression studies of variant MCAD proteins in E.coli -- Protein misfolding as a pathogenic mechanism in MCAD deficiency.…”
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