Search Results - "proteins"

  1. 521

    Matrix metalloproteinases in the central nervous system

    Published 2005
    Subjects: “…Extracellular matrix proteins.…”
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  2. 522
  3. 523

    Manual on feeding infants and young children / by Cameron, Margaret, 1921-

    Published 1983
    Subjects:
    Book
  4. 524
  5. 525

    The hyperphosphorylation of Tau and Alzheimer's disease by Goedert, Michel.spk

    Published 2010
    Table of Contents: “…Contents: Alzheimer's disease -- Extracellular plaques -- Intracellular tangles -- Tau filaments -- Tau hyperphosphorylation -- Other tauopathies -- Mouse models of tauopathy -- Protein kinase inhibitors -- Protein phosphatase activators.…”
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  6. 526

    SMAD phosphorylation and the TGF-beta pathway

    Published 2010
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  7. 527

    Nucleosome dynamics and remodeling

    Published 2007
    Table of Contents: “…Contents: Chromatin composition -- Nucleosome structure and function -- Dynamic properties inherent to nucleosomes -- Invasion of nucleosomes by gene regulatory proteins -- DNA binding proteins and enzymes -- ATP dependent remodeling factors -- Nucleosome relocation.…”
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  8. 528

    The principle of translation

    Published 2007
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  9. 529

    Chromatin and transcription

    Published 2007
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  10. 530

    Role and regulation of Cdk inhibitors in development and cancer

    Published 2009
    Table of Contents: “…Contents: Somatic cell cycle -- Rb pathway -- Positive regulation of G1 phase by phosphorylation of the retinoblastoma protein (Rb) by cyclin-dependent kinases (Cdks) -- Negative regulation of G1 by Cdk-inhibitory proteins (CKIs) -- Rb pathway is often disrupted in cancers -- Two families of CKIs, differential expression in embryogenesis and adult mouse tissues -- Loss of p18Ink4c and p19ink4d induces male sterility -- Loss of p19Ink4d induces progressive deafness after birth -- Loss of p19Ink4d and p27Kip1 induces re-entry of differentiated postmitotic neurons in cycle after birth -- Loss of Ink4c and Patched or p53 induces medulloblastoma, a pediatric tumor of the cerebellum -- CKIs are required for exit from and maintenance of cell cycle exit, or quiescence, a reversible state -- Loss of CKIs leads to sterility, deafness and cancers.…”
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  11. 531
  12. 532

    Protein and amino acid requirements in human nutrition report of a joint WHO/FAO/UNU expert consultation.

    Published 2007
    Subjects: “…Proteins Nutrition Requirements.…”
    An electronic book accessible through the World Wide Web; click to view
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  13. 533

    How can molecular chaperones repair damaged protein structures?

    Published 2021
    Table of Contents: “…Contents: The role of molecular chaperones -- Concept of polypeptide unfolding enzymes -- The RubisCO binding protein -- Molecular chaperones can proof-read the quality of protein folding -- Structure and function of Hsp70 -- The folding state of Hsp70 substrate -- Substrate targeting -- Entropic pulling -- J-domain cochaperones.…”
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  14. 534

    How can molecular chaperones repair damaged protein structures?

    Published 2021
    Table of Contents: “…Contents: The role of molecular chaperones -- Concept of polypeptide unfolding enzymes -- The RubisCO binding protein -- Molecular chaperones can proof-read the quality of protein folding -- Structure and function of Hsp70 -- The folding state of Hsp70 substrate -- Substrate targeting -- Entropic pulling -- J-domain cochaperones.…”
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  15. 535

    Protein and peptide folding, misfolding, and non-folding

    Published 2012
    Subjects: “…Protein folding.…”
    An electronic book accessible through the World Wide Web; click to view
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  16. 536

    RNA structure and folding : biophysical techniques and prediction methods /

    Published 2013
    Subjects: “…Protein folding.…”
    An electronic book accessible through the World Wide Web; click to view
    Electronic eBook
  17. 537

    Polycomb epigenetic mechanisms part 1 of 2, role of PcG complexes /

    Published 2017
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  18. 538

    Pathogenic mechanisms in prion disease

    Published 2014
    Table of Contents: “…Contents: Neurodegenerative diseases and protein misfolding disorders -- Prion disease in mice to study mechanisms of neurodegeneration due to misfolded protein accumulation -- Early prion neurodegenerative change is reversible: access to key neurotoxic pathways -- Critical change is loss of synaptic proteins due to activation of PERK branch of the unfolded protein response -- Modifying this pathway genetically is neuroprotective -- Modifying this pathway pharmacologically is neuroprotective -- Relevance of this pathway in other neurodegenerative diseases with misolded proteins e.g. …”
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  19. 539

    Roles for Hsp40 molecular chaperones in protein misfolding disease

    Published 2012
    Table of Contents: “…Contents: Protein aggregation in conformational disease -- The Hsp40 system -- The Hsp70 system -- Small oligomers, prion, yeast, Huntington's disease and protein triage -- Mechanisms for proteotoxicity -- Mechanisms for amyloid disease.…”
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  20. 540

    Role of calnexin and calreticulin in protein homeostasis within the endoplasmic reticulum

    Published 2012
    Table of Contents: “…Contents: Introduction to the endoplasmic reticulum -- Protein folding and quality control within the endoplasmic reticulum -- Asn-linked glycosylation and oligosaccharide processing -- Calnexin and calreticulin -- Structure and functions -- The Calnexin cycle -- Recognition of substrate polypeptide by calnexin and calreticulin -- The dual binding model -- Role of ERp57 in glycoprotein folding by calnexin and calreticulin -- Peptidyl prolyl isomerases within the endoplasmic reticulum -- ER-associated degradation -- Overview -- Recognition of misfolded glycoproteins -- The Glyco code of the ER.…”
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