Search Results - "glucose"

O-GlcNAc, an essential regulator of numerous cellular pathways

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O-GlcNAc, an essential regulator of numerous cellular pathways

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O-GlcNAc, an essential regulator of numerous cellular pathways

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Evolution of the diagnostic criteria for diabetes

Table of Contents: “…Contents: Description & symptoms of diabetes -- Criteria for screening and challenges -- Abbreviated history of diabetes diagnosis -- Glucose distribution (unimodal & bimodal) -- Cutoff values & mathematical manipulations -- Different criteria for diagnosis of diabetes & diabetes classifications -- WHO criteria for diabetes mellitus -- Relationship between FPG & 2hr value on OGTT -- Decisions of the expert committee -- Glycation -- The Pima Indians, Egyptian, and NHANES III studies -- Prevalence of retinopathy -- Oral glucose tolerance tests -- Quantitation of variation -- Using HbA1c levels to diagnose diabetes -- Expert committee on diagnosis of diabetes -- Screening for diabetes before and after 2010 ADA criteria and recommendations for diabetes diagnosis.…”
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Evolution of the diagnostic criteria for diabetes

Table of Contents: “…Contents: Description & symptoms of diabetes -- Criteria for screening and challenges -- Abbreviated history of diabetes diagnosis -- Glucose distribution (unimodal & bimodal) -- Cutoff values & mathematical manipulations -- Different criteria for diagnosis of diabetes & diabetes classifications -- WHO criteria for diabetes mellitus -- Relationship between FPG & 2hr value on OGTT -- Decisions of the expert committee -- Glycation -- The Pima Indians, Egyptian, and NHANES III studies -- Prevalence of retinopathy -- Oral glucose tolerance tests -- Quantitation of variation -- Using HbA1c levels to diagnose diabetes -- Expert committee on diagnosis of diabetes -- Screening for diabetes before and after 2010 ADA criteria and recommendations for diabetes diagnosis.…”
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Series

Evolution of the diagnostic criteria for diabetes

Table of Contents: “…Contents: Description & symptoms of diabetes -- Criteria for screening and challenges -- Abbreviated history of diabetes diagnosis -- Glucose distribution (unimodal & bimodal) -- Cutoff values & mathematical manipulations -- Different criteria for diagnosis of diabetes & diabetes classifications -- WHO criteria for diabetes mellitus -- Relationship between FPG & 2hr value on OGTT -- Decisions of the expert committee -- Glycation -- The Pima Indians, Egyptian, and NHANES III studies -- Prevalence of retinopathy -- Oral glucose tolerance tests -- Quantitation of variation -- Using HbA1c levels to diagnose diabetes -- Expert committee on diagnosis of diabetes -- Screening for diabetes before and after 2010 ADA criteria and recommendations for diabetes diagnosis.…”
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Managing hyperglycaemia in people with DM and CKD part 2, cardiovascular disease (CVD) involvement /

Table of Contents: “…Contents: The need to individualise glycaemic control in DKD -- The role of new agents in reducing development and progression of DKD -- The need for co-ordinated DM care in haemodialysis -- Logical and inappropriate combination blood glucose lowering therapies in DKD -- Safe management of DKD during acute illness.…”
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Managing hyperglycaemia in people with DM and CKD part 2, cardiovascular disease (CVD) involvement /

Table of Contents: “…Contents: The need to individualise glycaemic control in DKD -- The role of new agents in reducing development and progression of DKD -- The need for co-ordinated DM care in haemodialysis -- Logical and inappropriate combination blood glucose lowering therapies in DKD -- Safe management of DKD during acute illness.…”
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PI3K/AKT signaling In cancer

Table of Contents: “…Contents: PI3K/AKT signal transduction pathway -- Tumors with frequent dysregulation of PI3K signalling -- Modalities for inhibiting PI3K-AKT signaling currently in development -- AKT1/2 inhibitor (Merck) -- PI3K/AKT in breast cancer -- AKTi-1/2 inhibits AKT signaling and causes G1 arrest -- The Herceptin-resistant -- Effect of AKT inhibition on glucose levels -- Mutated elements in mitogenic signaling pathways in tumors -- Coexistent K-Ras or B-Raf mutation -- Genes encoding several components of mitogenic signaling pathways are mutationally activated in tumors -- Integrators of multiple signaling pathways (OR-gates) -- The effects of combined inhibition of AKT and MAPK on the phosphorylation of translational regulatory proteins -- Knockdown of 4EBP1 expression -- Feedback inhibition of insulin/IGF-1 signaling -- The concept of feedback -- Clinical effects of Rapamycin-like drugs -- mTORC1 vs. …”
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PI3K/AKT signaling In cancer

Table of Contents: “…Contents: PI3K/AKT signal transduction pathway -- Tumors with frequent dysregulation of PI3K signalling -- Modalities for inhibiting PI3K-AKT signaling currently in development -- AKT1/2 inhibitor (Merck) -- PI3K/AKT in breast cancer -- AKTi-1/2 inhibits AKT signaling and causes G1 arrest -- The Herceptin-resistant -- Effect of AKT inhibition on glucose levels -- Mutated elements in mitogenic signaling pathways in tumors -- Coexistent K-Ras or B-Raf mutation -- Genes encoding several components of mitogenic signaling pathways are mutationally activated in tumors -- Integrators of multiple signaling pathways (OR-gates) -- The effects of combined inhibition of AKT and MAPK on the phosphorylation of translational regulatory proteins -- Knockdown of 4EBP1 expression -- Feedback inhibition of insulin/IGF-1 signaling -- The concept of feedback -- Clinical effects of Rapamycin-like drugs -- mTORC1 vs. …”
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Series

PI3K/AKT signaling In cancer

Table of Contents: “…Contents: PI3K/AKT signal transduction pathway -- Tumors with frequent dysregulation of PI3K signalling -- Modalities for inhibiting PI3K-AKT signaling currently in development -- AKT1/2 inhibitor (Merck) -- PI3K/AKT in breast cancer -- AKTi-1/2 inhibits AKT signaling and causes G1 arrest -- The Herceptin-resistant -- Effect of AKT inhibition on glucose levels -- Mutated elements in mitogenic signaling pathways in tumors -- Coexistent K-Ras or B-Raf mutation -- Genes encoding several components of mitogenic signaling pathways are mutationally activated in tumors -- Integrators of multiple signaling pathways (OR-gates) -- The effects of combined inhibition of AKT and MAPK on the phosphorylation of translational regulatory proteins -- Knockdown of 4EBP1 expression -- Feedback inhibition of insulin/IGF-1 signaling -- The concept of feedback -- Clinical effects of Rapamycin-like drugs -- mTORC1 vs. …”
Get full text
Series