Search Results - "glucose"

The blood-brain barrier in Alzheimer's disease

Table of Contents: “…Contents: The blood-brain barrier -- Alzheimer's disease -- The blood-brain barrier in Alzheimer's disease -- Cerebral blood flow -- Glucose transport -- Aβ clearance -- Blood-brain barrier leakage -- Blood-brain barrier repair.…”
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The blood-brain barrier in Alzheimer's disease

Table of Contents: “…Contents: The blood-brain barrier -- Alzheimer's disease -- The blood-brain barrier in Alzheimer's disease -- Cerebral blood flow -- Glucose transport -- Aβ clearance -- Blood-brain barrier leakage -- Blood-brain barrier repair.…”
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O-GlcNAc, an essential regulator of numerous cellular pathways

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O-GlcNAc, an essential regulator of numerous cellular pathways

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O-GlcNAc, an essential regulator of numerous cellular pathways

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Physical activity, sedentary behaviour and diabetes

Table of Contents: “…Contents: Epidemiological thinking: physical activity & health -- Skeletal muscle, glucose disposal and muscle glucose uptake -- Recent advances in exercise and T2DM -- Exercise modality, combination training and rationale for strength, resistance training -- Exercise guidelines for T2DM (2006) -- Change in HbA1c -- "Stickiness" -- Community centre vs. home based resistance training -- Physical activity recommendations and public health -- The "sitting orientated" society -- Sedentary behaviours and mortality -- Movement and posture measurement devices -- Sedentary time and breaks (NHANES) -- Australian adults' overall daily behaviour patterns -- Rethinking the physical activity paradigm -- Pathophysiology of prolonged sitting -- Sedentary time and light-intensity activity -- How to reduce sitting time and guidance for prescribing exercise and physical activity promotion -- ACSM position stand.…”
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Physical activity, sedentary behaviour and diabetes

Table of Contents: “…Contents: Epidemiological thinking: physical activity & health -- Skeletal muscle, glucose disposal and muscle glucose uptake -- Recent advances in exercise and T2DM -- Exercise modality, combination training and rationale for strength, resistance training -- Exercise guidelines for T2DM (2006) -- Change in HbA1c -- "Stickiness" -- Community centre vs. home based resistance training -- Physical activity recommendations and public health -- The "sitting orientated" society -- Sedentary behaviours and mortality -- Movement and posture measurement devices -- Sedentary time and breaks (NHANES) -- Australian adults' overall daily behaviour patterns -- Rethinking the physical activity paradigm -- Pathophysiology of prolonged sitting -- Sedentary time and light-intensity activity -- How to reduce sitting time and guidance for prescribing exercise and physical activity promotion -- ACSM position stand.…”
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Physical activity, sedentary behaviour and diabetes

Table of Contents: “…Contents: Epidemiological thinking: physical activity & health -- Skeletal muscle, glucose disposal and muscle glucose uptake -- Recent advances in exercise and T2DM -- Exercise modality, combination training and rationale for strength, resistance training -- Exercise guidelines for T2DM (2006) -- Change in HbA1c -- "Stickiness" -- Community centre vs. home based resistance training -- Physical activity recommendations and public health -- The "sitting orientated" society -- Sedentary behaviours and mortality -- Movement and posture measurement devices -- Sedentary time and breaks (NHANES) -- Australian adults' overall daily behaviour patterns -- Rethinking the physical activity paradigm -- Pathophysiology of prolonged sitting -- Sedentary time and light-intensity activity -- How to reduce sitting time and guidance for prescribing exercise and physical activity promotion -- ACSM position stand.…”
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PI3K/AKT signaling In cancer

Table of Contents: “…Contents: PI3K/AKT signal transduction pathway -- Tumors with frequent dysregulation of PI3K signalling -- Modalities for inhibiting PI3K-AKT signaling currently in development -- AKT1/2 inhibitor (Merck) -- PI3K/AKT in breast cancer -- AKTi-1/2 inhibits AKT signaling and causes G1 arrest -- The Herceptin-resistant -- Effect of AKT inhibition on glucose levels -- Mutated elements in mitogenic signaling pathways in tumors -- Coexistent K-Ras or B-Raf mutation -- Genes encoding several components of mitogenic signaling pathways are mutationally activated in tumors -- Integrators of multiple signaling pathways (OR-gates) -- The effects of combined inhibition of AKT and MAPK on the phosphorylation of translational regulatory proteins -- Knockdown of 4EBP1 expression -- Feedback inhibition of insulin/IGF-1 signaling -- The concept of feedback -- Clinical effects of Rapamycin-like drugs -- mTORC1 vs. …”
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PI3K/AKT signaling In cancer

Table of Contents: “…Contents: PI3K/AKT signal transduction pathway -- Tumors with frequent dysregulation of PI3K signalling -- Modalities for inhibiting PI3K-AKT signaling currently in development -- AKT1/2 inhibitor (Merck) -- PI3K/AKT in breast cancer -- AKTi-1/2 inhibits AKT signaling and causes G1 arrest -- The Herceptin-resistant -- Effect of AKT inhibition on glucose levels -- Mutated elements in mitogenic signaling pathways in tumors -- Coexistent K-Ras or B-Raf mutation -- Genes encoding several components of mitogenic signaling pathways are mutationally activated in tumors -- Integrators of multiple signaling pathways (OR-gates) -- The effects of combined inhibition of AKT and MAPK on the phosphorylation of translational regulatory proteins -- Knockdown of 4EBP1 expression -- Feedback inhibition of insulin/IGF-1 signaling -- The concept of feedback -- Clinical effects of Rapamycin-like drugs -- mTORC1 vs. …”
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PI3K/AKT signaling In cancer

Table of Contents: “…Contents: PI3K/AKT signal transduction pathway -- Tumors with frequent dysregulation of PI3K signalling -- Modalities for inhibiting PI3K-AKT signaling currently in development -- AKT1/2 inhibitor (Merck) -- PI3K/AKT in breast cancer -- AKTi-1/2 inhibits AKT signaling and causes G1 arrest -- The Herceptin-resistant -- Effect of AKT inhibition on glucose levels -- Mutated elements in mitogenic signaling pathways in tumors -- Coexistent K-Ras or B-Raf mutation -- Genes encoding several components of mitogenic signaling pathways are mutationally activated in tumors -- Integrators of multiple signaling pathways (OR-gates) -- The effects of combined inhibition of AKT and MAPK on the phosphorylation of translational regulatory proteins -- Knockdown of 4EBP1 expression -- Feedback inhibition of insulin/IGF-1 signaling -- The concept of feedback -- Clinical effects of Rapamycin-like drugs -- mTORC1 vs. …”
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