Search Results - "Alcoholism"

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  1. 1

    Pancreatitis and Ca2+ signalling

    Published 2007
    Table of Contents: “…Contents: Normal Calcium signaling events in pancreatic acinar cells -- Hyperstimulation as a cause of sustained global elevation of the cytosolic [Calcium], Calcium-dependent vacuolization and Calcium-dependent protease activation -- Bile acids causing sustained global Calcium signals and necrosis -- Calcium-dependent necrosis evoked by non-oxidative alcohol metabolites (fatty acid ethyl esters and fatty acids) but not by alcohol or acetaldehyde -- The mechanism of action of fatty acid ethyl esters and fatty acids -- The mildly protective effect of coffee (caffeine) -- A model for the initiation of alcoholic pancreatitis.…”
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    Series (Calcium signaling I)
    Series (Calcium signaling II)
    Electronic Video
  2. 2

    Pancreatitis and Ca2+ signalling

    Published 2007
    Table of Contents: “…Contents: Normal Calcium signaling events in pancreatic acinar cells -- Hyperstimulation as a cause of sustained global elevation of the cytosolic [Calcium], Calcium-dependent vacuolization and Calcium-dependent protease activation -- Bile acids causing sustained global Calcium signals and necrosis -- Calcium-dependent necrosis evoked by non-oxidative alcohol metabolites (fatty acid ethyl esters and fatty acids) but not by alcohol or acetaldehyde -- The mechanism of action of fatty acid ethyl esters and fatty acids -- The mildly protective effect of coffee (caffeine) -- A model for the initiation of alcoholic pancreatitis.…”
    Get full text
    Series (Calcium signaling I)
    Series (Calcium signaling II)
    Electronic Video
  3. 3

    Pancreatitis and Ca2+ signalling

    Published 2007
    Table of Contents: “…Contents: Normal Calcium signaling events in pancreatic acinar cells -- Hyperstimulation as a cause of sustained global elevation of the cytosolic [Calcium], Calcium-dependent vacuolization and Calcium-dependent protease activation -- Bile acids causing sustained global Calcium signals and necrosis -- Calcium-dependent necrosis evoked by non-oxidative alcohol metabolites (fatty acid ethyl esters and fatty acids) but not by alcohol or acetaldehyde -- The mechanism of action of fatty acid ethyl esters and fatty acids -- The mildly protective effect of coffee (caffeine) -- A model for the initiation of alcoholic pancreatitis.…”
    Get full text
    Series (Calcium signaling I)
    Series (Calcium signaling II)
    Electronic Video